Flonase Information
What's inside Flonase
Flonase Active Ingredients: fluticasone propionate, details.| Flonase Dosages & Strengths | ||||
|---|---|---|---|---|
| Strength | Format | Route | Strength | Class |
| Flonase 0.05 mg/inh | spray | nasal | 1.0 inhalation | OTC |
Recent Flonase Side Effects
Posted by idreamofjeanne 3 days ago
It made me bloated, excitable, moody, depressed, angry and most of all, lost my sense of smell. Not to mention, 2 weeks afte...
Posted by italia2 2 months ago
I have a 3 year old that was taking Singulair until today after I read the horrible side effects that I was seeing in my chil...
Posted by catsnpaw 5 months ago
Flonase worked very well for my allergies for about 3 years then I developed a resistance to it. I can honestly say I don't t...
Flonase in the News
AMITYVILLE, NY--(BUSINESS WIRE)--Hi-Tech Pharmacal Co., Inc ... - Business Wire (press release)
Mr. Seltzer further commented, “We are very up-beat about our generic business due to our recent approval of generic Flonase®*, and more recently, ...
Mon Mar 10 08:05:07 -0400 2008
Hi-Tech Pharmacal says milder winter hurt drug sales - Newsday
In January, Hi-Tech won final approval from the US Food and Drug Administration for a new nasal spray, Flonase, and the company said in its news release ...
Mon Mar 10 09:09:08 -0400 2008
Don't marry him! - San Diego CityBEAT
Tonight, Sam and I stood in the kitchen, each ingesting two squirts per nostril of our own individually marked bottles of Flonase. ...
Tue Mar 04 21:57:24 -0500 2008
Flonase Chemical Information
fluticasone propionate - The propionate salt form of fluticasone, a synthetic trifluorinated glucocorticoid receptor agonist with antiallergic, antiinflammatory and antipruritic effects. Binding and activation of the glucocorticoid receptor results in the activation of lipocortin that in turn inhibits cytosolic phospholipase A2, which triggers cascade of reactions involved in synthesis of inflammatory mediators, such as prostaglandins and leukotrienes. Secondly, mitogen-activated protein kinase (MAPK) phosphatase 1 is induced, thereby leads to dephosphorylation and inactivation of Jun N-terminal kinase directly inhibiting c-Jun mediated transcription. Finally, transcriptional activity of nuclear factor (NF)-kappa-B is blocked, thereby inhibits the transcription of cyclooxygenase 2, which is essential for prostaglandin production.
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