Dyazide Information

What's inside Dyazide

Recent Dyazide Side Effects

Posted by jcgrier24 2 months ago
I had to go off Yaz due to it causing my blood pressure to skyrocket, causing horrific acne on my face, excessive mood swings...

Posted by redkitty2001 3 months ago
I had been on Yaz for about 1 1/2 years with little to no problems until the last couple of months I began to get severely de...

Posted by ltreadway 4 months ago
Our daughter spent her 21st birthday in ICU, Sunday the 13th. I took her to the ER with shortness of breath, chest pain and ...

Dyazide in the News

No cure, many treatments for Meniere’s - Enid News & Eagle
... the pressure caused by excess fluids in the inner ear, initial treatment may include a low-salt diet along with a diuretic medication such as dyazide. ...
Mon Jul 02 20:05:26 -0400 2007

No secondary assistance and which represents open. - Journal lycéen
Further studies working life dyazide delivery situations dyclonine penetratid replicat loads. The nature department for overall award fatal. ...
Sun Jun 17 17:12:44 -0400 2007

Cops accuse man of sending bulk in mail - Penn Live
Courts documents say the drugs include Nandrolone, Winstrol, Provirion, Apavrin, Fitropin, Dyazide, Dufine, Broncoiterol, and HGC. ...
Thu May 24 06:01:48 -0400 2007

Dyazide Chemical Information

hydrochlorothiazide - The hydrogenated derivative of chlorothiazide, a thiazide diuretic with antihypertensive and anti-urolithic effects. This agent binds to the electroneutral Na-K-Cl cotransporter (NKCC) and thereby impairs Na+, K+ and Cl- reabsorption on the luminal membrane of the early segment in the distal convoluted tubule in the kidney. This leads to an increase in urinary excretion of sodium, chloride, potassium, bicarbonate and water subsequently reducing plasma and extracellular fluid volume leading to a reduction in blood pressure. Hydrochlorothiazide also decreases urinary calcium and uric acid excretion by direct action on the distal tubule.

triamterene - A pteridine derivative with potassium-sparing diuretic property. Triamterene blocks the sodium-potassium exchange pump (Na-K-ATPase) in the luminal membrane of principal cells in the late distal tubule, cortical collecting tubule and collecting duct in the kidney. This reversible inhibition of the electrogenic sodium transport decreases the lumen-negative transepithelial potential difference and thus reduces the driving force for K+ movement into the tubular lumen resulting in the inhibition of sodium reabsorption in exchange for K+ and H+.